132.8
[1]. Särnstrand B, et al. N,N-Diacetyl-L-cystine-the disulfide dimer of N-acetylcysteine-is a potent modulator of contact sensitivity/delayed type hypersensitivity reactions in rodents. J Pharmacol Exp Ther. 1 Mar;288(3):117-8.
[2]. Wågberg M, et al. N,N-diacetyl-L-cystine (DiNAC), the disulphide dimer of N-acetylcysteine, inhibits atherosclerosis in WHHL rabbits: evidence for immunomodulatory agents as a new approach to prevent atherosclerosis. J Pharmacol Exp Ther. 2001 Oct;2(1):7-82.
[3]. B S?rnstrand, et al. N,N-Diacetyl-L-cystine-the disulfide dimer of N-acetylcysteine-is a potent modulator of contact sensitivity/delayed type hypersensitivity reactions in rodents. J Pharmacol Exp Ther. 1 Mar;288(3):117-8.
[]. Knut Pettersson, et al. The antiatherogenic effect of DiNAC: experimental findings supporting immunomodulation as a new treatment for atherosclerosis related diseases. Cardiovasc Drug Rev. 2003 Summer;21(2):11-32.
[5]. Knut Pettersson, et al. The new oral immunomodulating drug DiNAC induces brachial artery vasodilatation at rest and during hyperemia in hypercholesterolemic subjects, likely by a nitric oxide-dependent mechanism. Atherosclerosis. 2008 Jan;1(1):275-282.
H302-H315-H31-H335
P21-P2-P270-P271-P280-P302+P352-P30+P30-P305+P351+P338-P330-P32+P3-P03+P233-P05-P501